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Exosomal lncRNA TUG1 derived from human urine-derived stem cells attenuates renal ischemia/reperfusion injury by interacting with SRSF1 to regulate ASCL4-mediated ferroptosis
Taken together, these findings suggest that NSBSO-induced GSH depletion and synthesis inhibition can elevate intracellular ROS, thereby activating the caspase family, leading to GSDME cleavage and cell pyroptosis
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Retrieved from (licensed in all 50 states) Bean, M
The crosstalk between autophagy and ferroptosis is implicated in the molecular mechanisms of liver injury induced by various exogenous toxins
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